I am a gentle man, filled with love but fighting as a digital soldier for truth and global peace. Forgiveness is the way forward for the horrific crimes that have been perpetrated against us. We don’t need revenge, what we need is retribution and redemption.
Juxtapose the word Ivermectin with redemption or retribution, it makes you stop and think… what’s the big deal with Ivermectin? Why does that drug matter? The reason that Ivermectin matters is that the Big-pharma cabal and all their minions lied about this drug, they knew full-well that it works wonders. Murder is a word that comes to mind, revenge is another.
To withhold the truth is the sin of omission, perhaps revenge is the price to pay. An eye for an eye, you decide.
Doctors and organizations that advocated for ivermectin, and were silenced during the pandemic – Dr. Tess Lawrie’s British Ivermectin Recommendation Development (BIRD) to California pulmonologist Roger Seheult and many others, were shut down online with the same unilateral abruptness social media platforms apply to hate speech or death threats.
Dr. Sabine Hazan, a gastroenterologist and CEO of a genetic sequencing laboratory called ProGenaBiome in Ventura, California, was blindsided. She got involved with ivermectin when she was pulling out the stops for Covid-19 patients. Dr. Hazan says. When patients got really sick, she tried everything, treating off-label with a number of drugs in combination, including ivermectin.
Eventually, she ended up taking it upon herself to run clinical trials with repurposed, off-patent drugs like ivermectin and hydroxychloroquine. Dr. Sabine Hazan shared her findings, only to have her video removed by Youtube.
- Ivermectin is an inhibitor of the COVID-19 causative virus (SARS-CoV-2) in vitro.•
- A single treatment able to effect ~5000-fold reduction in virus at 48 h in cell culture.•
- Ivermectin is FDA-approved for parasitic infections, and therefore has a potential for repurposing.•
- Ivermectin is widely available, due to its inclusion on the WHO model list of essential medicines.
Ivermectin is an effective treatment for COVID-19. Treatment is more effective when used early.
From bacteria and plants to novel anti-parasite therapies
After decades of limited progress in developing durable therapies for parasitic diseases, the discoveries by this year’s Laureates (2015 Nobel Prize in Physiology or Medicine) radically changed the situation.
Satoshi Ōmura, a Japanese microbiologist and expert in isolating natural products, focused on a group of bacteria, Streptomyces, which lives in the soil and was known to produce a plethora of agents with antibacterial activities (including Streptomycin discovered by Selman Waksman, Nobel Prize 1952). Equipped with extraordinary skills in developing unique methods for large-scale culturing and characterization of these bacteria, Ōmura isolated new strains of Streptomyces from soil samples and successfully cultured them in the laboratory. From many thousand different cultures, he selected about 50 of the most promising, with the intent that they would be further analyzed for their activity against harmful microorganisms
The 2015 Nobel Prize in Physiology and Medicine was awarded to William C. Campbell and Satoshi Ömura for their discoveries leading to ivermectin. In addition to its extraordinary efficacy against parasitic diseases, ivermectin continues to offer new clinical applications due to its ability to be repurposed to treat new classes of diseases. Beyond its invaluable therapeutic role in onchocerciasis and strongyloidiasis, an increasing body of evidence points to the potential of ivermectin as an antiviral agent.
If that were cause enough for retribution, consider that in 2005 Dr. Anthony Fauci authored a paper touting the therapeutic benefits of Chloroquine. Even the title of the peer reviewed scientific paper suggests that there was a natural solution for the corona virus:
Severe acute respiratory syndrome (SARS) is caused by a newly discovered coronavirus (SARS-CoV). No effective prophylactic or post-exposure therapy is currently available.
We report, however, that chloroquine has strong antiviral effects on SARS-CoV infection of primate cells. These inhibitory effects are observed when the cells are treated with the drug either before or after exposure to the virus, suggesting both prophylactic and therapeutic advantage. In addition to the well-known functions of chloroquine such as elevations of endosomal pH, the drug appears to interfere with terminal glycosylation of the cellular receptor, angiotensin-converting enzyme 2. This may negatively influence the virus-receptor binding and abrogate the infection, with further ramifications by the elevation of vesicular pH, resulting in the inhibition of infection and spread of SARS CoV at clinically admissible concentrations.
Chloroquine is effective in preventing the spread of SARS CoV in cell culture. Favorable inhibition of virus spread was observed when the cells were either treated with chloroquine prior to or after SARS CoV infection. In addition, the indirect immunofluorescence assay described herein represents a simple and rapid method for screening SARS-CoV antiviral compounds.
Keywords: severe acute respiratory syndrome coronavirus, chloroquine, inhibition, therapy